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The Throwback Collection of Morgellons SpecimensIn the first three years I suffered with Morgellons disease I was totally on my own. All I knew was that I had something on and in my body that I could see and feel but my doctors were trying to tell me I was having delusions of parasites. They also told me the ulcerous sores on my body were the results of self excoriation. I made all of the classic moves like bringing in fibers to show the them. The humiliating treatment I received from the medical profession only succeeded in making me very angry and fueled my great determination to prove them wrong. I thought that I was the only person going through this misery and didn't learn for several years that other had these same symptoms. In the beginning I was looking for something with a logical and biological explanation. I was not into any sort of conspiracy theories. I simply thought I would get a microscope and study what was on my body. I believed that with enough studying and looking through the scope that I would identify what disease I had from a known database of recognized diseases. I began a 3 year study of all fungus, cyanobacteria, chytrids, rotifirs, algae or anything else I could learn about that might be a clue to this mystery. Eventually I looked at the Internet and typed some symptoms into a search engine. I was amazed and excited that I found Mary Leitao's Morgellons website. I contacted Mary and started working with her. I asked her about the name “Morgellons”. She had taken it from a 1690 monograph by Sir Thomas Brown who described a condition in children that included copious growth of hair on their backs. I can't say that I agreed with her choice to use that particular name for this illness but I supposed it sounded more medically correct than what I have been calling it which was “the crap.” During the time I worked with Mary she was able to get some of my Morgellons samples into a lab at the University of Pittsburgh. They underwent testing using Fourier Transform Infrared Spectroscopy. They were found to be hollow, auto fluorescent and to contain cellulose but no chitin. From my earlier study of mycology I knew that the absence chitin ruled out any fungus since all fungus contains chitin. At that time I got a letter from Mary and with some micrograph images taken by the lab. Due to a computer assault from an evil anti-Morgellons website I no longer have copies of these. Apparently the samples were quite a sensation at the lab and could not be readily identified. There were to be further studies done and frozen cross sections for further testing had already prepared by the lab. Shortly thereafter I learned that the University had told Mary they had no time to work on it. This was surprising since it was a unique material with many unanswered questions and this was, after all, a University where I would hope knowledge would be valued. I began to wonder why the Morgellons research had been dumped like a hot potato. Several years later more fiber samples from my body were determined to be made of HDPE ( high-density polyethylene) by another researcher who paid to have them tested at several accredited labs. There are differences in the fibers since some are round and some are more ribbon-shaped. There may also be a difference if a fiber directly enters the body from the environment or if it is a second or more generation product of self replication that occurs in the body.
In later years it was apparent to me that the bizarre nature of my Morgellons findings were unnatural and likely from a man-made source. It was not a pleasant journey as I began to "wake up" to what was happening in this altered reality. Much to my dismay, all roads carried me to the same conclusion and still do. The pathogens in my body came from a bio-lab and have the hand of man written all over them. It occurred to me that if these pathogens were being bioengineered in a lab that they were made of multiple components and that the mutated material might reproduce and intermittently send out a batch of identifiable debris much like the original genetics. I call this type of debris
“throwbacks”. The multi-component part of my theory has proven to be true. Read my paper entitled: "Revelations From a Man Who Helped Design Morgellons Disease" (RE: Dr Gwen Scott and her admissions from a government scientist) It is my theory that when new life forms or designer diseases are created in bio-labs using the processes of gene splicing, mutation, bioengineering or nanotechnology that components of these mixtures can self replicate with varied results. Components such as dictyolstelium discoidium, insect genes, and other pathogens will continue to make some new mutations in each generation of self replication. I believe that some of these mutations "throw back" to expressions similar to the original materials used in the lab created mixture. Specimens from Morgellons disease tissue can sometimes be identified as nearly identical to the original components used in this biological witches brew before the mutation process. It is not unlike finding a child with flaming red hair in a family of people who do not have this characteristic. The information in this paper is only my personal theory but it is the best explanation I can find to make sense of all of the strange artifacts that come from people with Morgellons disease. It is also why I think that some of these strange findings occur most frequently in the early phases of the disease and do not repeat as often in later years of the disease. I will later discuss a strange cellular slime mold known as dictyostelium discoidium. It is all about using the genetic characteristics of this cellular slime mold to produce mutations in a bio-lab. I believe it is one of many parts of the Morgellons story. Most of my “throwback” findings only appeared in the beginning of the disease when I had multiple lesions and things were at their worst. That was about 7 years ago. I had tentatively identified some of the debris as members of 3 varieties of the Oomycota phylum of pseudo fungus. There are over 500 varieties and many of them can have ill effects of humans, animals and plants. The varieties of oomycota can produce diseases like pythiosis, lagenidosis, rhinosporidosis, downy mildew, blue tobacco mold, the fish disease saprolegnia (Ich) and the infamous phytophthora infestans that had killed millions in the Irish Potato Famine by blighting the spuds. I had also found bizarre mutations of earthworms and other material that looked like dictyostelium discoidium. The reason that the dictyostelium discoidium is important is because this cellular slime mold has unique properties that lend itself to mutating other substances. These eukaryotic microorganisms have a simplistic genetic makeup and produces what are know as chemotaxis. Dictyostelium is utilized in many labs to specifically mutate other material. Dictyostelium has an amoeba form along with many other stages of varied shapes and configurations as the individual cells emerge into groupings that look like larger single entities. “Dictyostelium amoebae grow as separate, independent cells but interact to form multicellular structures when challenged by adverse conditions such as starvation. Up to 100,000 cells signal each other by releasing the chemoattractant cAMP and aggregate together by chemotaxis to form a mound that is surrounded by an extracellular matrix. Processes depend on cell-cell communication in Dictyostelium. Many of the underlying molecular and cellular processes appear to have arisen in primitive precursor cells and to have remained fundamentally unchanged throughout evolution. Basic processes of development such as differential cell sorting, pattern formation, stimulus-induced gene expression, and cell-type regulation are common to Dictyostelium. It is used in gene research as well as other uses” (dictybase.org). Even if you have never before heard of dictyostelium discoidium you may be quite impressed to go to this huge website that is well funded and part of the Human Genome Project and NIH. This cellular slime mold is a major player in many aspects of medicine and cell research. It is a good bit of information to have for future reference. I encourage everyone to take a look at photos and videos of this substance at http://dictybase.org/ The many configurations of Dictyostelium: Each shape is comprised of hundreds to thousands of single motile amoeba cells acting in unison to form each of these configurations. Take a good look at the photos below. There is a definite presence of Dictyostelium in the tissue from Morgellons victims. This can only mean that our disease is man made in a bio-lab. The pathogens we have are no doubt a mixture of the Dictyostelium and other pathogens that were purposely mutated with it for a deliberate purpose. Dictyostelium is not normally a pathogen to humans. It feeds on bacteria and under normal circumstances a human is not a good host due to lack of bacteria and a body temperature that exceeds it's ideal gestation temperature of 72-77 degrees Fahrenheit. It is obvious that the dictyostelium has been mutated to even be able survive on the human body.
A lawn of starved Dictyostelium amoeba cells is imaged using phase contrast microscopy. Cells signal via spiral waves of cAMP, and population territories form with a fruiting body in the center of each.
Dictyostelium Discoidium Life Cycle cAMP receptor 1 (cAR1) of Dictyostelium couples to the G protein G2 to mediate activation of chemotaxis, and cell aggregation.
Amoeba formation as found in Morgellons
Unusual antenna formation on amoeba
Spiral geometry of a signal transmitter cAMP in an amoeba population (Dictyostelium discoideum) leads to chemotactic movements of cells in direction of the spiral core. This is the signaling phase where cells communicate and aggregate.
Streaming pattern
Jan's Photo Tentacles (dictyostelium)
The left photo shows the beginning and ending phases of dictyostelium metamorphosis. The photo on the right is the translation of mutated dictyostelium as found inside a lesion on the human body. The formation of the dictyostelium component continues to mutate and vary. This one throws back to to the original morphology. Other specimens take the tentacle form and do not elongate and produce the bulb on the end. The basic forms are apparent but the actual function of bulb on the top producing sporulation is now gone. ~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~ From: "Texas Medical Center News" Vol. 21, No. 18 October 1, 1999 Genome Studies of Slug Might be Applicable to Humans "Dictyostelium consists of only six chromosomes and approximately 8,000 genes. The DNA in those genes comprises 34 million pairs of chemicals called bases that contain instructions for the role each gene plays. " "By "knocking out" or deleting a gene and observing the effect of the mutation on Dictyostelium's development, Dr. Kuspa can determine the gene's function. It takes only a month to generate a mutation in Dictyostelium, but in a more complicated genome, such as that of a mouse, the process can require six months to a year. "We have developed methods that enable one laboratory researcher to mutate 100 Dictyostelium genes within a month," Dr. Kuspa says."
~~~~~~~~~~~~~~~~~~~~~~~~~~~ Here are some of the “throwbacks” I found in my own tissue from the phylum oomycota (o-o-my-CO-ta) 3 photos of saprolegnia zoosporangium.
These are not the original coloration as shown except for small image on lower left fibers were dark blue, bulbs on the ends of fibers were brown/red. The bulbs (zoosporangium) would fall off as a separate component. I only found this formation twice, about 5 years ago.
The photo below is not from my body but I did find a specimen exactly like this. I was able to get a drop of the clear sticky liquid that came out of a lesion onto a slide. At a magnification of 350x I found a tiny but uniquely distinct shape on the slide. I could not then identify it but I described it to Mary Leitao as a clear snake skin with tiny round circles in it. I was sure this slide would be a breakthrough for the research but unfortunately that did not happen.
I did not have a microscope that I could take a digital image with at that time. I did have a scientific microscope. I sent this slide to Mary Leitao who is a trained biologist. She looked at it and acknowledged the specimen on the slide. She forwarded that slide and many others to Dr. William Harvey who was then in private practice. The slides were said to have been “lost” during a move by his office. Formerly Dr. Harvey had worked for NASA full time and at that time was still the Chairman of their Educational Advisory Board. I must admit that I still question what happened to my slides. I later identified the missing specimen myself as Dictyuchus from a web photo.
All of the varieties of oomycota I have mentioned thus far are related to the fish disease known as Ich or Saprolegnia. I do not believe that Morgellons victims have a pure strain of this disease but I do believe that some of the genetics were used from this disease in the bioengineered mixture.
I believe that other varieties of the oomycota family have been put into the mix as well. I have previously found this star fish shape in my lesions. I know that others have found them as well
Top photo: Oomycota Peronospora Tabacina or blue tobacco
mold.
This is the last of the oomycota from my body. I have found a few of these grooved, coffee bean shaped pieces in the past but this one was found only a few weeks ago. This zoospore is Phytophthora infestans, the villain in the Irish potato famine. This is a motile zoospore powered by two flagellum. One is a long whiplash flagella and a shorter one is the shorter tinsel flagella. When these zoospores are threatened they encyst and can hide out in their encapsulated form for a very long while. Sometimes they release the flagellum and in other instances they retract the flagellum and they become encysted as well. The whiplash flagella has a shape similar to a worm-like structure I often find which I call the motile strand. The composition of oomycota is cellulose and glucans which store mycolaminarin and is a form of sugar. If glucans and cellulose are added to the human body could the results of this addition = excess sugars and carbohydrates = diabetes? I wonder since I have recently been diagnosed with it and there is an epidemic of it in this country. I will search for answers and write a follow up. Web photos of phytophthora infestans compared to Morgellons motile strand.
This is what I found in a lesion on my body.
According to Wikepedia Phytophthora infestans Potato blight was one of more than 17 agents that the United States researched as potential biological weapons before the nation suspended its biological weapons program.[15][dead link] France, Canada, USA and the Soviet Union also researched P. infestans as a biological weapon in the 1940s and 50s.[16] Do we really believe that this former candidate as a biological weapon was discontinued by the Government? What a coincidence that it is turning up in the tissue of Morgellons victims. What I do know thus far is that some of the motile (moving) strands found on my body were tested with Raman Spectroscopy in the labs at SUNY Stony Brook. The results were surprising to me at the time since I expected the fibers to be silicon or HDPE (high density polyethylene) due to previous results of other types of Morgellons samples that had undergone professional lab analysis. These strands were made of an indeterminate kind of polysaccharide (sugar substance- polymer and other unknowns). More testing was to be done on this material but this was not possible since the researcher, quite suddenly, was no longer allowed to use their lab. Funny how any progress on Morgellons research seems to end suddenly as soon as the research results are posted on the Internet. I wondered how many sugar/polysaccharide based organisms could produce motile strands with independent movement. Once again I could only think of the zoospores of the polysaccharide based oomycota and the whiplash flagella of pythium insidiosum. There are some chytrids that produce motile zoospores but not in long stranded fibers. All roads lead me back to my original conclusion of oomycota. The researcher, Biologist Mark Darrah, nicknamed these Sugar Snakes. The coloration varies with these strands. They are sometimes a white crystal-laden translucent coloration and vary to brown or black as well as striped mixtures of several colors. The high magnification that was used by Mark Darrah clearly shows a striped banding on the structures. Mark shined a laser light on one of the strands and it literally melted. There is a deformity in it's surface at the top left of the photo below. Here is what he stated to me in about this: “This is a pimple that formed when the laser light hit it demonstrating heat sensitivity.” To see more of Mark's work go to: Mark Darrah Research There are 7 pages of his lab photos at this site.
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